Download e-book for iPad: Advances in Chronic Kidney Disease 2010: 12th International by P. Kotanko, N. W. Levin, C. Ronco

By P. Kotanko, N. W. Levin, C. Ronco

ISBN-10: 3805593406

ISBN-13: 9783805593403

The complaints of the overseas convention on Dialysis, prepared via the Renal study Institute, manhattan, are released each year in "Blood Purification". one of the themes mentioned within the present quantity are: the opportunity of erythropoiesis-stimulating brokers for tissue safety; remedy of high blood pressure in dialysis sufferers; the capability position of gelsolin in dialysis-related protein-energy losing; salt limit in dialysis sufferers; blood-pressure-independent results of aldosterone on nonepithelial tissues; development of the process heart problems by way of modulating immune reaction; the potential for intestinal healing interventions to minimize uremic toxin new release and absorption; the 'obesity paradox' in dialysis sufferers; the importance of endothelial progenitor cells and endothelial vesicles in CKD; the administration of calcium stability in persistent dialysis sufferers; and, the position of peripheral ultrafiltration within the administration of acute decompensated middle failure. final yet no longer least, numerous papers are devoted to neighborhood citrate anticoagulation instead to systemic heparin anticoagulation. concentrating on fresh advancements in operational components, know-how, biology of uremia and epidemiology, this book is a beneficial reference instrument for either clinicians and simple investigators occupied with CKD.

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Additional info for Advances in Chronic Kidney Disease 2010: 12th International Conference on Dialysis, New Orleans, La., January 2010. Special Issue: Blood Purification 2010, Vol. 29, No. 2

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1159/000245635 Published online: January 8, 2010 Aldosterone in Uremia – Beyond Blood Pressure Eberhard Ritz a Nadezda Koleganova b a Nierenzentrum and b Department of Pathology, Ruperto Carola University Heidelberg, Heidelberg, Germany Key Words Aldosterone ؒ Salt ؒ Blood pressure ؒ Endothelial cell function ؒ Dialysis Abstract Aldosterone was in the past considered only as a prohypertensinogenic agent. e. causing blood pressure elevation as a result of salt retention, aldosterone has numerous blood-pressure-independent actions on nonepithelial tissue.

E. digitalis-analogous endogenous steroids), increased blood pressure [14] and caused bloodpressure-independent cardiomyopathy and accelerated progression of kidney disease [15]. Recent studies of Oberleithner et al. [16] have carried this one step further: raising the extracellular sodium concentration from 135 to 160 mmol/l caused a gradual increase in endothelial stiffness, reduced nitric oxide production and caused endothelial cell shape change. The presence of aldosterone was permissive for this effect.

First, two classes of the renin-angiotensin system (angiotensin-converting enzyme inhibitor and angiotensin II receptor blockers, antihypertensive agents with proven anti-inflammatory activity) have been tested in clinical trials on a dialysis population [38, 39]. The results of a randomized trial using foStinghen /Bucharles /Riella /Pecoits-Filho sinopril [38] showed a slight benefit for this agent in comparison to placebo. In another randomized trial with a small number of patients [39], candesartan significantly reduced cardiovascular events and mortality in patients on chronic maintenance hemodialysis.

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Advances in Chronic Kidney Disease 2010: 12th International Conference on Dialysis, New Orleans, La., January 2010. Special Issue: Blood Purification 2010, Vol. 29, No. 2 by P. Kotanko, N. W. Levin, C. Ronco


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